BCL6 BTB‐specific inhibitor reversely represses T‐cell activation, Tfh cells differentiation, and germinal center reaction in vivo
Author:
Affiliation:
1. The Wistar Institute Philadelphia PA USA
2. Gilead Sciences, Inc. 333 Lakeside Dr Foster City CA 94404
3. Department of Pharmaceutical Sciences University of Maryland School of Pharmacy Baltimore MD USA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/eji.202049150
Reference32 articles.
1. BCL6 Evolved to Enable Stress Tolerance in Vertebrates and Is Broadly Required by Cancer Cells to Adapt to Stress
2. BCL6-mediated Attenuation of DNA Damage Sensing Triggers Growth Arrest and Senescence through a p53-dependent Pathway in a Cell Context-dependent Manner
3. Integrated biochemical and computational approach identifies BCL6 direct target genes controlling multiple pathways in normal germinal center B cells
4. Bcl6 Protein Expression Shapes Pre-Germinal Center B Cell Dynamics and Follicular Helper T Cell Heterogeneity
5. Bcl6 Mediates the Development of T Follicular Helper Cells
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