iNKT cells with high PLZF expression are recruited into the lung via CCL21‐CCR7 signaling to facilitate the development of asthma tolerance in mice

Author:

Feng Xintong12ORCID,Zhao Caiqi3,Li Ling3,Feng Jingjing12,He Wei12,Shi Tianyun12,Li Na12,Jie Zhijun12,Su Xiao3

Affiliation:

1. Department of Pulmonary and Critical Care Medicine, Shanghai Fifth People's Hospital Fudan University Shanghai China

2. Center of Community‐Based Health Research Fudan University Shanghai China

3. Unit of Respiratory Infection and Immunity, Institute Pasteur of Shanghai Chinese Academy of Sciences Shanghai China

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference46 articles.

1. Type 2 inflammation in asthma: present in most, absent in many (vol 15, pg 57, 2015);Fahy J. V.;Nat. Rev. Immunol.,2015

2. The role of immune tolerance in asthma pathogenesis;Tournoy K. G.;Curr. Allergy Asthma Rep.,2006

3. Elevated faecal 12,13‐diHOME concentration in neonates at high risk for asthma is produced by gut bacteria and impedes immune tolerance;Levan S. R.;Nat. Microbiol.,2019

4. Mechanisms of oral tolerance;Tordesillas L.;Clin. Rev. Allergy Immunol.,2018

5. Oral tolerance: therapeutic implications for autoimmune diseases;Faria A. M.;Clin. Dev. Immunol.,2006

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