Expression of a carboxy terminally truncated Stat5 with no transactivation domain in the mammary glands of transgenic mice inhibits cell proliferation during pregnancy, delays onset of milk secretion, and induces apoptosis upon involution
Author:
Publisher
Wiley
Subject
Cell Biology,Developmental Biology,Genetics
Reference44 articles.
1. Functionally Distinct Isoforms of STAT5 Are Generated by Protein Processing
2. Real-time imaging of ?-lactoglobulin-targeted luciferase activity in the mammary glands of transgenic mice
3. Synthesis and secretion of caseins by the mouse mammary gland: Production and characterization of new polyclonal antibodies
4. Elements Within the -Lactoglobulin Gene Inhibit Expression of Human Serum Albumin cDNA and Minigenes in Transfected Cells but Rescue Their Expression in the Mammary Gland of Transgenic Mice
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1. STAT5 confers lactogenic properties in breast tumorigenesis and restricts metastatic potential;Oncogene;2022-10-19
2. H2AX Promoter Demethylation at Specific Sites Plays a Role in STAT5-Induced Tumorigenesis;Journal of Mammary Gland Biology and Neoplasia;2020-08-03
3. Pregnancy reprograms the enhancer landscape of mammary epithelial cells and alters the response to cMYC-driven oncogenesis;2019-05-20
4. An Epigenetic Memory of Pregnancy in the Mouse Mammary Gland;Cell Reports;2015-05
5. An N-terminal splice variant of human Stat5a that interacts with different transcription factors is the dominant form expressed in invasive ductal carcinoma;Cancer Letters;2014-04
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