Müller glial cells inhibit proliferation of retinal endothelial cells via TGF-β2 and Smad signaling

Author:

Yafai Yousef1,Iandiev Ianors1,Lange Johannes2,Unterlauft Jan Darius1,Wiedemann Peter1,Bringmann Andreas1,Reichenbach Andreas3,Eichler Wolfram1

Affiliation:

1. Department of Ophthalmology and Eye Hospital; University of Leipzig; Leipzig Germany

2. Norwegian Centre for Movement Disorders; Stavanger University Hospital; Stavanger Norway

3. Paul Flechsig Institute of Brain Research, University of Leipzig; Leipzig Germany

Funder

Deutsche Forschungsgemeinschaft

Ernst und Berta Grimmke Stiftung, D�sseldorf, Germany

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Reference55 articles.

1. Immunological mechanisms in the pathogenesis of diabetic retinopathy;Adamis;Semin Immunopathol,2008

2. Hypoxic regulation of vascular endothelial growth factor in retinal cells;Aiello;Arch Ophthalmol,1995

3. Vascular endothelial growth factor is present in glial cells of the retina and optic nerve of human subjects with nonproliferative diabetic retinopathy;Amin;Invest Ophthalmol Vis Sci,1997

4. An activated form of transforming growth factor beta is produced by cocultures of endothelial cells and pericytes;Antonelli-Orlidge;Proc Natl Acad Sci USA,1989

5. Effects of hypoxia on glial cell expression of angiogenesis-regulating factors VEGF and TGF-β;Behzadian;Glia,1998

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