Role of hypoxia and cAMP in the transdifferentiation of human fetal cardiac fibroblasts: Implications for progression to scarring in autoimmune-associated congenital heart block-Reference-Cited by-同舟云学术

Role of hypoxia and cAMP in the transdifferentiation of human fetal cardiac fibroblasts: Implications for progression to scarring in autoimmune-associated congenital heart block

Published:2007-12 Issue:12 Volume:56 Page:4120-4131
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis Rheum

Author:

Clancy Robert M.,Zheng Ping,O'Mahony Marguerita,Izmirly Peter,Zavadil Jiri,Gardner Lawrence,Buyon Jill P.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference39 articles.

1. Risk of congenital complete heart block in newborns of mothers with anti-Ro/SSA antibodies detected by counterimmunoelectrophoresis: a prospective study of 100 women;Brucato;Arthritis Rheum,2001

2. Immunohistologic evidence supports apoptosis, IgG deposition, and novel macrophage/fibroblast crosstalk in the pathologic cascade leading to congenital heart block;Clancy;Arthritis Rheum,2004

3. Transdifferentiation of cardiac fibroblasts: a fetal factor in anti-SSA/Ro-SSB/La antibody-mediated congenital heart block;Clancy;J Immunol,2002

4. Anti-SSA/Ro and anti-SSB/La autoantibodies bind the surface of apoptotic fetal cardiocytes and promote secretion of TNF-α by macrophages;Miranda-Carus;J Immunol,2000

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