Lipopolysaccharide‐induced hypothalamic inflammation in cancer cachexia‐anorexia is amplified by tumour‐derived prostaglandin E2
Author:
Affiliation:
1. Nutritional Biology, Division of Human Nutrition Wageningen University Wageningen The Netherlands
2. Papé Family Pediatric Research Institute Oregon Health & Science University (OHSU) Portland OR USA
Publisher
Wiley
Subject
Physiology (medical),Orthopedics and Sports Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcsm.13093
Reference40 articles.
1. Cancer cachexia: understanding the molecular basis
2. Increased hypothalamic serotonin turnover in inflammation-induced anorexia
3. The central role of hypothalamic inflammation in the acute illness response and cachexia
4. The role of hypothalamic inflammation, the hypothalamic–pituitary–adrenal axis and serotonin in the cancer anorexia–cachexia syndrome
5. Muscle Atrophy in Response to Cytotoxic Chemotherapy Is Dependent on Intact Glucocorticoid Signaling in Skeletal Muscle
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1. Exploring heterogeneity: a dive into preclinical models of cancer cachexia;American Journal of Physiology-Cell Physiology;2024-08-01
2. Defining and Addressing Research Priorities in Cancer Cachexia through Transdisciplinary Collaboration;Cancers;2024-06-27
3. Central neural mechanisms of cancer cachexia;Current Opinion in Supportive & Palliative Care;2024-05-15
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