AP‐1 and Mitf interact with NFATc1 to stimulate cathepsin K promoter activity in osteoclast precursors
Author:
Affiliation:
1. Division of Geriatrics and Palliative Medicine, Jacobs School of Medicine and Biomedical Sciences University at Buffalo Buffalo New York
2. Veterans Affairs Western New York Healthcare System Research Service Buffalo New York
Funder
Veteran Affairs BLR&D
Indian Trail Foundation
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.28504
Reference41 articles.
1. The role of cathepsin K in normal bone resorption
2. Osteoclast differentiation and gene regulation
3. Clinical and animal research findings in pycnodysostosis and gene mutations of cathepsin K from 1996 to 2011
4. Dental Abnormalities Caused by Novel Compound Heterozygous CTSK Mutations
5. Impaired bone resorption in cathepsin K-deficient mice is partially compensated for by enhanced osteoclastogenesis and increased expression of other proteases via an increased RANKL/OPG ratio
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