NF-κB2 Controls the Migratory Activity of Memory T Cells by Regulating Expression of CXCR4 in a Mouse Model of Sjögren's Syndrome
Author:
Affiliation:
1. Tokushima University Graduate School of Biomedical Sciences; Tokushima Japan
2. Garvan Institute of Medical Research; Darlinghurst New South Wales Australia
Funder
Japan Society for the Promotion of Science
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.40230/fullpdf
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3. Chemokines determine local lymphoneogenesis and a reduction of circulating CXCR4+ T and CCR7 B and T lymphocytes in thyroid autoimmune diseases;Armengol;J Immunol,2003
4. Among CXCR3 chemokines, IFN-γ-inducible protein of 10 kDa (CXC chemokine ligand (CXCL) 10) but not monokine induced by IFN-γ (CXCL9) imprints a pattern for the subsequent development of autoimmune disease;Christen;J Immunol,2003
5. Chemokine receptor trio: CXCR3, CXCR4 and CXCR7 crosstalk via CXCL11 and CXCL12;Singh;Cytokine Growth Factor Rev,2013
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