The Mitochondrial Permeability Transition Pore, the c-Subunit of the F1FoATP Synthase, Cellular Development, and Synaptic Efficiency-Reference-Cited by-同舟云学术

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The Mitochondrial Permeability Transition Pore, the c-Subunit of the F1FoATP Synthase, Cellular Development, and Synaptic Efficiency

Published:2015-09-25 Issue: Volume: Page:31-64
ISSN:
Container-title:The Functions, Disease-Related Dysfunctions, and Therapeutic Targeting of Neuronal Mitochondria
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Author:

Jonas Elizabeth A.,Porter George A.,Beutner Gisela,Mnatsakanyan Nelli,Alavian Kambiz N.

Publisher

John Wiley & Sons, Inc

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/9781119017127.ch2/fullpdf

Reference189 articles.

1. Targeted polyphosphatase expression alters mitochondrial metabolism and inhibits calcium-dependent cell death;Abramov;Proc Natl Acad Sci U S A,2007

2. The Bcl-2 apoptotic switch in cancer development and therapy;Adams;Oncogene,2007

3. Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase;Alavian;Nat Cell Biol,2011

4. An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore;Alavian;Proc Natl Acad Sci U S A,2014a

5. An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore;Alavian;Proc Natl Acad Sci U S A,2014b

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