Interleukin-23/Th17 pathways and inflammatory bowel disease
Author:
Publisher
Oxford University Press (OUP)
Subject
Gastroenterology,Immunology and Allergy
Reference182 articles.
1. Interleukin-23 drives innate and T cell–mediated intestinal inflammation
2. STAT3 and NF-κB Signal Pathway Is Required for IL-23-Mediated IL-17 Production in Spontaneous Arthritis Animal Model IL-1 Receptor Antagonist-Deficient Mice
3. IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2–dependent mechanisms with implications for psoriasis pathogenesis
4. The Orphan Nuclear Receptor RORγt Directs the Differentiation Program of Proinflammatory IL-17+ T Helper Cells
5. Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitis
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