Evidence for a molecular mechanism of teratogenicity of SB-236057, a 5-HT1B receptor inverse agonist that alters axial formation-Reference-Cited by-同舟云学术

Evidence for a molecular mechanism of teratogenicity of SB-236057, a 5-HT1B receptor inverse agonist that alters axial formation

Published:2004-10 Issue:10 Volume:70 Page:789-807
ISSN:1542-0752
Container-title:Birth Defects Research Part A: Clinical and Molecular Teratology
language:en
Short-container-title:Birth Defect Res A

Author:

Augustine-Rauch Karen A.,Zhang Qin J.,Leonard Jennifer L.,Chadderton Antony,Welsh Michael J.,Rami Harshad K.,Thompson Mervyn,Gaster Laramie,Wier Patrick J.

Publisher

Wiley

Subject

Developmental Biology,Embryology,General Medicine,Pediatrics, Perinatology, and Child Health

Reference41 articles.

1. R-esp1, a rat homologue ofDrosophilaGroucho, is differentially expressed after optic nerve crush and mediates NGF-induced survival of PC12 cells

2. Antisense attenuation ofWnt-1 andWnt-3a expression in whole embryo culture reveals roles for these genes in craniofacial, spinal cord, and cardiac morphogenesis

3. Linking Notch signaling, chromatin remodeling, and T-cell leukemogenesis

4. Quantitation of rat embryonic development in vitro: A morphological scoring system

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