NFAT1 deficit and NFAT2 deficit attenuate EAE via different mechanisms-Reference-Cited by-同舟云学术

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NFAT1 deficit and NFAT2 deficit attenuate EAE via different mechanisms

Published:2015-02-19 Issue:5 Volume:45 Page:1377-1389
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Dietz Lena¹,Frommer Friederike¹²,Vogel Anna-Lena¹,Vaeth Martin¹,Serfling Edgar¹,Waisman Ari²,Buttmann Mathias³,Berberich-Siebelt Friederike¹⁴

Affiliation:

1. Institute of Pathology; University of Wuerzburg; Wuerzburg Germany

2. Institute for Molecular Medicine; University Medical Center of the Johannes Gutenberg; University of Mainz; Mainz Germany

3. Department of Neurology; University of Wuerzburg; Wuerzburg Germany

4. Comprehensive Cancer Center Mainfranken; University of Wuerzburg; Wuerzburg Germany

Funder

Federal Ministry for Education and Research

German Research Foundation

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/eji.201444638/fullpdf

Reference53 articles.

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2. Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis;Beecham;Nat. Genet.,2013

3. Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis;Sawcer;Nature,2011

4. Pathophysiology of multiple sclerosis;Korn;J. Neurol.,2008

5. Evidence for cytokine dysregulation in multiple sclerosis: peripheral blood mononuclear cell production of pro-inflammatory and anti-inflammatory cytokines during relapse and remission;Hollifield;Autoimmunity,2003

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