IL-33 attenuates the development of experimental autoimmune uveitis-Reference-Cited by-同舟云学术

IL-33 attenuates the development of experimental autoimmune uveitis

Published:2014-10-18 Issue:11 Volume:44 Page:3320-3329
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Barbour Mark¹,Allan Debbie¹,Xu Heping²,Pei Cheng¹,Chen Mei²,Niedbala Wanda³,Fukada Sandra Y.³,Besnard Anne-Galle³,Alves-Filho Jose C.³,Tong Xiaoguang¹,Forrester John V.⁴⁵⁶,Liew Foo Yew³⁷⁸,Jiang Hui-Rong¹

Affiliation:

1. Strathclyde Institute of Pharmacy and Biomedical Sciences; University of Strathclyde; Glasgow UK

2. Centre for Experimental Medicine; Queen's University Belfast; Belfast UK

3. Institute of Infection, Immunity and Inflammation; University of Glasgow; Glasgow UK

4. Institute of Medical Science; University of Aberdeen; UK

5. Centre for Ophthalmology and Visual Science; The University of Western Australia; Australia

6. Centre for Experimental Immunology; Lions Eye Institute; Nedlands Australia

7. CEGMR; King Abdulaziz University; Jeddah Saudi Arabia

8. Institute of Biology and Medical Sciences; Soochow University; Suzhou China

Funder

University of Strathclyde

MRC

Welcome Trust

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference46 articles.

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3. Sialoadhesin promotes the inflammatory response in experimental autoimmune uveoretinitis;Jiang;J. Immunol.,2006

4. Either a Th17 or a Th1 effector response can drive autoimmunity: conditions of disease induction affect dominant effector category;Luger;J. Exp. Med.,2008

5. Inhibitory peptide analogs derived from a major uveitogenic epitope protect from antiretinal autoimmunity by inducing type 2 and regulatory T cells;Cortes;J. Leukoc. Biol.,2008

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