The expression of mouse CLEC-2 on leucocyte subsets varies according to their anatomical location and inflammatory state

Author:

Lowe Kate L.1,Navarro-Núñez Leyre1,Bénézech Cécile2,Nayar Saba3,Kingston Bethany L.34,Nieswandt Bernhard5,Barone Francesca3,Watson Steve P.1,Buckley Christopher D.3,Desanti Guillaume E.3

Affiliation:

1. Centre for Cardiovascular Sciences; University of Birmingham; Birmingham UK

2. MRC Centre for Immune Regulation; University of Birmingham; Birmingham UK

3. Centre for Translational Inflammation Research; Rheumatology Research Group; University of Birmingham; Birmingham UK

4. Medical School; University of Oxford; Oxford UK

5. Department of Experimental Biomedicine; University Hospital, University of Würzburg; Würzburg Germany

Funder

Arthritis Research UK

British Heart Foundation

Medical Research Council

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference52 articles.

1. RANKL/RANK - from bone physiology to breast cancer;Sigl;Cytokine Growth Factor Rev.,2014

2. The physiological and pathophysiological roles of platelet CLEC-2;Navarro-Nunez;Thromb. Haemost.,2013

3. T1 alpha protein is developmentally regulated and expressed by alveolar type I cells, choroid plexus, and ciliary epithelia of adult rats;Williams;Am. J. Respir. Cell Mol. Biol.,1996

4. Podoplanin, novel 43-kd membrane protein of glomerular epithelial cells, is down-regulated in puromycin nephrosis;Breiteneder-Geleff;Am. J. Pathol.,1997

5. Characterization of an antigenic determinant preferentially expressed by type I epithelial cells in the murine thymus;Farr;J. Histochem. Cytochem.,1992

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