miR-148a is upregulated by Twist1 and T-bet and promotes Th1-cell survival by regulating the proapoptotic gene Bim-Reference-Cited by-同舟云学术

miR-148a is upregulated by Twist1 and T-bet and promotes Th1-cell survival by regulating the proapoptotic gene Bim

Published:2015-01-22 Issue:4 Volume:45 Page:1192-1205
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Haftmann Claudia¹,Stittrich Anna-Barbara¹²,Zimmermann Jakob¹,Fang Zhuo¹³,Hradilkova Kristyna¹,Bardua Markus¹,Westendorf Kerstin¹,Heinz Gitta A.¹⁴,Riedel René¹,Siede Julia¹⁵,Lehmann Katrin¹,Weinberger Esther E.¹⁵,Zimmel David¹⁵,Lauer Uta⁵,Häupl Thomas⁵,Sieper Joachim⁵,Backhaus Marina⁵,Neumann Christian¹,Hoffmann Ute¹⁵,Porstner Martina⁶,Chen Wei⁷,Grün Joachim R¹,Baumgrass Ria¹,Matz Mareen⁸,Löhning Max¹⁵,Scheffold Alexander⁵,Wittmann Jürgen⁶,Chang Hyun-Dong¹,Rajewsky Nikolaus³,Jäck Hans-Martin⁶,Radbruch Andreas¹,Mashreghi Mir-Farzin¹

Affiliation:

1. Deutsches Rheuma-Forschungszentrum Berlin (DRFZ); an institute of the Leibniz AssociationBerlin; Berlin Germany

2. Institute for Systems Biology; Seattle, WA USA

3. Laboratory of Systems Biology of Gene Regulatory Elements; Max-Delbrück-Center for Molecular Medicine; Berlin Germany

4. Institute of Molecular Immunology; Helmholtz Zentrum München; Munich Germany

5. Department of Rheumatology and Clinical Immunology; Charité-Universitätsmedizin Berlin; Berlin Germany

6. Division of Molecular Immunology; Friedrich-Alexander University Erlangen Nürnberg; Erlangen Germany

7. Laboratory of Novel sequencing technology; Function and Medical Genomics; Max-Delbrück-Center for Molecular Medicine; Berlin Germany

8. Department of Nephrology; Charité-Universitätsmedizin; Berlin Germany

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference53 articles.

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4. In-vitro-induced Th17 cells fail to induce inflammation in vivo and show an impaired migration into inflamed sites;Janke;Eur. J. Immunol.,2010

5. Loss of T-bet, but not STAT1, prevents the development of experimental autoimmune encephalomyelitis;Bettelli;J. Exp. Med.,2004

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