WISP1 silencing confers protection against epithelial–mesenchymal transition of renal tubular epithelial cells in rats via inactivation of the wnt/β‐catenin signaling pathway in uremia-Reference-Cited by-同舟云学术

WISP1 silencing confers protection against epithelial–mesenchymal transition of renal tubular epithelial cells in rats via inactivation of the wnt/β‐catenin signaling pathway in uremia

Published:2018-12-17 Issue:6 Volume:234 Page:9673-9686
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chen Yuan‐Zhen¹,Sun Dan‐Qin²,Zheng Yi³⁴,Zheng Guang‐Kuai¹,Chen Rong‐Quan²,Lin Mei¹,Huang Lian‐Fang¹,Huang Cong¹,Song Dan¹,Wu Ben‐Qing⁵^ORCID

Affiliation:

1. Department of Nephrology University of Chinese Academy of Sciences Shenzhen Hospital Shenzhen China

2. Department of Nephrology Wuxi No. 2 People's Hospital, Nanjing Medical University Wuxi China

3. Central Laboratory, University of Chinese Academy of Sciences Shenzhen Hospital Shenzhen China

4. Central Laboratory, Huadu District People's Hospital, Southern Medical University Guangzhou China

5. Children's Medical Center, University of Chinese Academy of Sciences Shenzhen Hospital Shenzhen China

Funder

Knowledge Innovation Program of Shenzhen Innovation Committee

Knowledge Innovation Program of Guangzhou Innovation Committee

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27654

Reference54 articles.

1. Reduced hydration status characterized by disproportionate elevation of blood urea nitrogen to serum creatinine among the patients with cerebral infarction

2. A homozygous mutation in INVS causing juvenile nephronophthisis with abnormal reactivity of the Wnt/beta-catenin pathway

3. WNT1 inducible signaling pathway protein 1 (WISP1): A novel mediator linking development and disease

4. β-Catenin Causes Renal Dysplasia via Upregulation ofTgfβ2andDkk1

5. Blood Urea Nitrogen/Creatinine Ratio Identifies a High-Risk but Potentially Reversible Form of Renal Dysfunction in Patients With Decompensated Heart Failure

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