Upregulation of microRNA‐424 relieved diabetic nephropathy by targeting Rictor through mTOR Complex2/Protein Kinase B signaling-Reference-Cited by-同舟云学术

Upregulation of microRNA‐424 relieved diabetic nephropathy by targeting Rictor through mTOR Complex2/Protein Kinase B signaling

Published:2019-01-13 Issue:7 Volume:234 Page:11646-11653
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Wang Guofeng¹^ORCID,Yan Yongxin¹,Xu Ning¹,Hui Yuan¹,Yin Dong¹

Affiliation:

1. Department of endocrinology The Affiliated Lianyungang Hospital of Xuzhou Medical University, The First People's Hospital of Lianyungang, The Affiliated Hospital of Kangda College of Nanjing Medical University Lianyungang Clinical College of Nanjing Medical University Lianyungang China

Funder

Lianyungang health bureau research project

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27822

Reference26 articles.

1. Ellagic acid, an NF-κB inhibitor, ameliorates renal function in experimental diabetic nephropathy

2. Distinguishing diabetic nephropathy from other causes of glomerulosclerosis: an update

3. miR‐424 protects PC‐12 cells from OGD‐induced injury by negatively regulating MKP‐1;Cao Y. X.;European Review for Medical and Pharmacological Sciences,2018

4. Improvements in the Management of Diabetic Nephropathy

5. Thioredoxin-interacting protein regulates lipid metabolism via Akt/mTOR pathway in diabetic kidney disease

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