Traumatically injured astrocytes release a proteomic signature modulated by STAT3-dependent cell survival-Reference-Cited by-同舟云学术

Traumatically injured astrocytes release a proteomic signature modulated by STAT3-dependent cell survival

Published:2015-12-19 Issue:5 Volume:64 Page:668-694
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Levine Jaclynn¹,Kwon Eunice¹,Paez Pablo²,Yan Weihong³,Czerwieniec Gregg³,Loo Joseph A.³⁴⁵,Sofroniew Michael V.⁶,Wanner Ina-Beate¹

Affiliation:

1. Semel Institute for Neuroscience and Human Behavior, David Geffen School of Medicine; University of California Los Angeles; Los Angeles California

2. Department of Pharmacology and Toxicology, Hunter James Kelly Research Institute, School of Medicine and Biomedical Sciences; SUNY, University at Buffalo, NYS Center of Excellence; Buffalo, New York

3. Department of Chemistry and Biochemistry; University of California Los Angeles; Los Angeles California

4. Department of Biological Chemistry, David Geffen School of Medicine; University of California; Los Angeles California

5. UCLA/DOE Institute for Genomics and Proteomics, University of California; Los Angeles California

6. Department of Neurobiology, David Geffen School of Medicine; University of California Los Angeles; Los Angeles California

Funder

National Institutes of Health

Craig H. Neilsen Foundation (82776 to IBW)

Dr. Miriam and Sheldon G. Adelson Medical Research Foundation

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Reference91 articles.

1. Stretch-induced injury alters mitochondrial membrane potential and cellular ATP in cultured astrocytes and neurons;Ahmed;J Neurochem,2000

2. Vulnerability of central neurons to secondary insults after in vitro mechanical stretch;Arundine;J Neurosci,2004

3. Mechanical cell injury;Barbee;Ann N Y Acad Sci,2005

4. The fate of glucose during the period of decreased metabolism after fluid percussion injury: A 13C NMR study;Bartnik;J Neurotrauma,2007

5. Mechanism of S100 protein-dependent inhibition of glial fibrillary acidic protein (GFAP) polymerization;Bianchi;Biochim Biophys Acta,1994

Cited by 52 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Proteomic Characterization of Changes in Mouse Brain Cortex Protein Expression at Different Post-Mortem Intervals: A Preliminary Study for Forensic Biomarker Identification;International Journal of Molecular Sciences;2024-08-10

2. An exploratory study of the damage markers NfL, GFAP, and t-Tau, in cerebrospinal fluid and other findings from a patient cohort enriched for suspected autoimmune psychiatric disease;Translational Psychiatry;2024-07-24

3. Characterisation of premature cell senescence in Alzheimer’s disease using single nuclear transcriptomics;Acta Neuropathologica;2024-05-02

4. NF-κB and JAK/STAT Signaling Pathways as Crucial Regulators of Neuroinflammation and Astrocyte Modulation in Spinal Cord Injury;Cells;2024-03-26

5. Reactive Astrocytes and Emerging Roles in Central Nervous System (CNS) Disorders;Cold Spring Harbor Perspectives in Biology;2024-02-05