Astrocyte sodium signaling and the regulation of neurotransmission-Reference-Cited by-同舟云学术

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Astrocyte sodium signaling and the regulation of neurotransmission

Published:2015-11-14 Issue:10 Volume:64 Page:1655-1666
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Kirischuk Sergei¹,Héja László²,Kardos Julianna²,Billups Brian³

Affiliation:

1. University Medical Center of the Johannes Gutenberg University Mainz, Institute of Physiology; Mainz Germany

2. Institute of Organic Chemistry, Research Centre for Natural Sciences, Hungarian Academy of Sciences; Budapest Hungary

3. Eccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University; Acton ACT Australia

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Reference135 articles.

1. External Ca(2+)-independent release of neurotransmitters;Adam-Vizi;J Neurochem,1992

2. Reversal or reduction of glutamate and GABA transport in CNS pathology and therapy;Allen;Pflugers Arch,2004

3. Depolarization-induced release of glycine and beta-alanine from plasma membrane vesicles derived from rat brain synaptosomes;Aragon;Biochim Biophys Acta,1988

4. GlyT1 and GlyT2 in brain astrocytes: Expression, distribution and function;Aroeira;Brain Struct Funct,2014

5. BDNF, via truncated TrkB receptor, modulates GlyT1 and GlyT2 in astrocytes;Aroeira;Glia,2015

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