EGFR/PPARδ/HSP90 pathway mediates cancer cell metabolism and chemoresistance-Reference-Cited by-同舟云学术

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EGFR/PPARδ/HSP90 pathway mediates cancer cell metabolism and chemoresistance

Published:2020-11-08 Issue:3-4 Volume:122 Page:394-402
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Gou Qian¹²³,Zhang Wenbo³,Xu Ying³,Jin Jianhua¹⁴,Liu Qian¹⁴,Hou Yongzhong¹³^ORCID,Shi Juanjuan³

Affiliation:

1. Department of Oncology, The Affiliated Wujin Hospital Jiangsu University Changzhou Jiangsu China

2. School of Medicine Jiangsu University Zhenjiang Jiangsu China

3. School of Life Science Jiangsu University Zhenjiang Jiangsu China

4. Department of Oncology The Wujin Clinical College of Xuzhou Medical University Xuzhou Jiangsu China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.29868

Reference44 articles.

1. PPARgamma E3 ubiquitin ligase regulates MUC1‐C oncoprotein stability;Hou Y;Oncogene,2013

2. PPARγ is an E3 ligase that induces the degradation of NFκB/p65

3. Peroxisome-proliferator-activated receptors and cancers: complex stories

4. PPARγ against Tumors by Different Signaling Pathways

5. Peroxisome proliferator-activated receptors (PPARs) are potential drug targets for cancer therapy

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2. Mitogen-Activated Protein Kinase and Exploratory Nuclear Receptor Crosstalk in Cancer Immunotherapy;International Journal of Molecular Sciences;2023-09-26

3. Unraveling the Role of Peroxisome Proliferator-Activated Receptor β/Δ (PPAR β/Δ) in Angiogenesis Associated with Multiple Myeloma;Cells;2023-03-25

4. Harnessing mitochondrial metabolism and drug resistance in non-small cell lung cancer and beyond by blocking heat-shock proteins;Drug Resistance Updates;2022-12

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