Hypothermia inhibits translocation of CaM kinase II and PKC-?, ?, ? isoforms and fodrin proteolysis in rat brain synaptosome during ischemia-reperfusion-Reference-Cited by-同舟云学术

Hypothermia inhibits translocation of CaM kinase II and PKC-?, ?, ? isoforms and fodrin proteolysis in rat brain synaptosome during ischemia-reperfusion

Published:2002-02-27 Issue:5 Volume:67 Page:664-669
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Harada Kazuki,Maekawa Tsuyoshi,Tsuruta Ryosuke,Kaneko Tadashi,Sadamitsu Daikai,Yamashima Tetsumori,Yoshida Ken-ichi

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference50 articles.

1. Ischemia-Induced Translocation of Ca2+/Calmodulin-Dependent Protein Kinase II: Potential Role in Neuronal Damage

2. Transient cerebral ischemia decreases calcium/ calmodulin-dependent protein kinase II immunoreactivity, but not mRNA levels in the gerbil hippocampus

3. Postischemic Administration of AK275, a Calpain Inhibitor, Provides Substantial Protection against Focal Ischemic Brain Damage

4. The spectrin skeleton: from red cells to brain.

5. Regulation of type-II calmodulin kinase: Functional implications

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