Impaired intracellular trafficking of sodium‐dependent vitamin C transporter 2 contributes to the redox imbalance in Huntington’s disease-Reference-Cited by-同舟云学术

Impaired intracellular trafficking of sodium‐dependent vitamin C transporter 2 contributes to the redox imbalance in Huntington’s disease

Published:2020-08-04 Issue:1 Volume:99 Page:223-235
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J Neurosci Res

Author:

Covarrubias‐Pinto Adriana¹,Parra Alejandra V.¹,Mayorga‐Weber Gonzalo¹,Papic Eduardo¹,Vicencio Isidora¹,Ehrenfeld Pamela²³,Rivera Francisco J.²³⁴⁵^ORCID,Castro Maite A.¹²⁶^ORCID

Affiliation:

1. Instituto de Bioquímica y Microbiología Facultad de Ciencias Universidad Austral de Chile Valdivia Chile

2. Center for Interdisciplinary Studies on the Nervous System (CISNe) Universidad Austral de Chile Valdivia Chile

3. Instituto de Anatomía, Histología y Patología, Facultad de Medicina Universidad Austral de Chile Valdivia Chile

4. Institute of Molecular Regenerative Medicine Paracelsus Medical University Salzburg Austria

5. Spinal Cord Injury and Tissue Regeneration Center Salzburg (SCI‐TReCS) Salzburg Austria

6. Janelia Research CampusHHMI Ashburn VA USA

Funder

Consejo Nacional de Innovación, Ciencia y Tecnología

Fondo Nacional de Desarrollo Científico y Tecnológico

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24693

Reference74 articles.

1. A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington’s disease in mice

2. Huntingtin has a membrane association signal that can modulate huntingtin aggregation, nuclear entry and toxicity

3. A prospective evaluation of cognitive decline in early Huntington's disease: Functional and radiographic correlates

4. Regional distribution of ascorbate and 3,4-dihydroxyphenylacetic acid (DOPAC) in rat striatum

5. Fast transport and retrograde movement of huntingtin and HAP 1 in axons

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