Activation of Akt–mTORC1 signalling reverts cancer‐dependent muscle wasting-Reference-Cited by-同舟云学术

Activation of Akt–mTORC1 signalling reverts cancer‐dependent muscle wasting

Published:2021-11-06 Issue:1 Volume:13 Page:648-661
ISSN:2190-5991
Container-title:Journal of Cachexia, Sarcopenia and Muscle
language:en
Short-container-title:J cachexia sarcopenia muscle

Author:

Geremia Alessia¹²,Sartori Roberta¹,Baraldo Martina¹²,Nogara Leonardo¹²,Balmaceda Valeria¹,Dumitras Georgia Ana¹²,Ciciliot Stefano¹,Scalabrin Marco¹²,Nolte Hendrik³,Blaauw Bert¹²^ORCID

Affiliation:

1. Veneto Institute of Molecular Medicine (VIMM) Padua Italy

2. Department of Biomedical Sciences University of Padua Padua Italy

3. Max Planck Institute for Biology of Ageing Cologne Germany

Funder

Associazione Italiana per la Ricerca sul Cancro

Università degli Studi di Padova

French Muscular Dystrophy Association

Publisher

Wiley

Subject

Physiology (medical),Orthopedics and Sports Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcsm.12854

Reference40 articles.

1. Cancer cachexia: understanding the molecular basis

2. Definition and classification of cancer cachexia: an international consensus

3. Molecular Mechanisms of Skeletal Muscle Hypertrophy

4. Comparative Analysis of Muscle Hypertrophy Models Reveals Divergent Gene Transcription Profiles and Points to Translational Regulation of Muscle Growth through Increased mTOR Signaling

5. Inducible activation of Akt increases skeletal muscle mass and force without satellite cell activation

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