AKT controls protein synthesis and oxidative metabolism via combined mTORC1 and FOXO1 signalling to govern muscle physiology-Reference-Cited by-同舟云学术

AKT controls protein synthesis and oxidative metabolism via combined mTORC1 and FOXO1 signalling to govern muscle physiology

Published:2021-11-09 Issue:1 Volume:13 Page:495-514
ISSN:2190-5991
Container-title:Journal of Cachexia, Sarcopenia and Muscle
language:en
Short-container-title:J cachexia sarcopenia muscle

Author:

Jaiswal Natasha¹,Gavin Matthew¹,Loro Emanuele²³,Sostre‐Colón Jaimarie¹,Roberson Paul A.⁴,Uehara Kahealani¹,Rivera‐Fuentes Nicole¹,Neinast Michael¹⁵,Arany Zoltan¹⁵,Kimball Scot R.⁴,Khurana Tejvir S.²³,Titchenell Paul M.¹²^ORCID

Affiliation:

1. Institute for Diabetes, Obesity, and Metabolism Perelman School of Medicine at the University of Pennsylvania Philadelphia PA USA

2. Department of Physiology Perelman School of Medicine at the University of Pennsylvania Philadelphia PA USA

3. Penn Muscle Institute, Department of Physiology Perelman School of Medicine at the University of Pennsylvania Philadelphia PA USA

4. Department of Cellular and Molecular Physiology Penn State College of Medicine Hershey PA USA

5. Cardiovascular Institute Perelman School of Medicine at the University of Pennsylvania Philadelphia PA USA

Funder

University of Pennsylvania

National Institutes of Health

Publisher

Wiley

Subject

Physiology (medical),Orthopedics and Sports Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcsm.12846

Reference50 articles.

1. Mechanisms of muscle atrophy and hypertrophy: implications in health and disease

2. AKT/PKB Signaling: Navigating the Network

3. Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways

4. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

5. ATP Citrate Lyase Improves Mitochondrial Function in Skeletal Muscle

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