Roles for H + /K + ‐ ATPase and zinc transporter 3 in cAMP ‐mediated lysosomal acidification in bafilomycin A1 ‐treated astrocytes-Reference-Cited by-同舟云学术

Roles for H + /K + ‐ ATPase and zinc transporter 3 in cAMP ‐mediated lysosomal acidification in bafilomycin A1 ‐treated astrocytes

Published:2020-12-14 Issue:5 Volume:69 Page:1110-1125
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Lee Huikyong¹,Koh Jae‐Young¹²^ORCID

Affiliation:

1. Neural Injury Laboratory, Biomedical Research Center, Asan Institute for Life Sciences, Asan Medical Center University of Ulsan College of Medicine Seoul South Korea

2. Department of Neurology, Asan Medical Center University of Ulsan College of Medicine Seoul South Korea

Funder

National Research Foundation of Korea

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23952

Reference48 articles.

1. Expression and regulation of H+K+ATPase in lysosomes of epithelial cells of the adult rat epididymis

2. Misrouting of v-ATPase subunit V0a1 dysregulates lysosomal acidification in a neurodegenerative lysosomal storage disease model

3. Neurotoxic astrocytes express the d-serine synthesizing enzyme, serine racemase, in Alzheimer's disease

4. The Ras/cAMP/Protein Kinase A Pathway Regulates Glucose-dependent Assembly of the Vacuolar (H+)-ATPase in Yeast

5. Kinetic Study of the Antiport Mechanism of an Escherichia coli Zinc Transporter, ZitB

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