Notch3 and DeltaB maintain Müller glia quiescence and act as negative regulators of regeneration in the light‐damaged zebrafish retina-Reference-Cited by-同舟云学术

Notch3 and DeltaB maintain Müller glia quiescence and act as negative regulators of regeneration in the light‐damaged zebrafish retina

Published:2020-09-23 Issue:3 Volume:69 Page:546-566
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Campbell Leah J.¹,Hobgood Joshua S.¹,Jia Meng¹,Boyd Patrick¹,Hipp Rebecca I.¹,Hyde David R.¹^ORCID

Affiliation:

1. Department of Biological Sciences, Center for Zebrafish Research, and the Center for Stem Cells and Regenerative Medicine, Galvin Life Science Center University of Notre Dame Notre Dame Indiana USA

Funder

National Eye Institute

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23912

Reference80 articles.

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3. Late-Stage Neuronal Progenitors in the Retina Are Radial Muller Glia That Function as Retinal Stem Cells

4. Notch–Delta signaling is required for spatial patterning and Müller glia differentiation in the zebrafish retina

5. Müller cells in the healthy and diseased retina

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