Investigating oligodendrocyte connexins: Heteromeric interactions between Cx32 and mutant or wild‐type forms of Cx47 do not contribute to or modulate gap junction function-Reference-Cited by-同舟云学术

Investigating oligodendrocyte connexins: Heteromeric interactions between Cx32 and mutant or wild‐type forms of Cx47 do not contribute to or modulate gap junction function

Published:2021-04-09 Issue:8 Volume:69 Page:1882-1896
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Abrams Charles K.¹²^ORCID,Flores‐Obando Rafael E.²,Dungan Gabriel D.¹,Cherepanova Elina²,Freidin Mona M.¹²^ORCID

Affiliation:

1. Department of Neurology and Rehabilitation University of Illinois at Chicago College of Medicine Chicago Illinois USA

2. Department of Neurology SUNY Downstate Medical Center Brooklyn New York USA

Funder

Muscular Dystrophy Association

National Multiple Sclerosis Society

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23999

Reference63 articles.

1. Evolutionary analyses of gap junction protein families

2. Diseases of connexins expressed in myelinating glia

3. Pathogenesis of X-Linked Charcot-Marie-Tooth Disease: Differential Effects of Two Mutations in Connexin 32

4. Functional Requirement for a Highly Conserved Charged Residue at Position 75 in the Gap Junction Protein Connexin 32

5. A new mutation in GJC2 associated with subclinical leukodystrophy

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