The protein kinase C super-family member PKN is regulated by mTOR and influences differentiation during prostate cancer progression

Author:

Yang Chun-Song1,Melhuish Tiffany A.12,Spencer Adam1,Ni Li1,Hao Yi12,Jividen Kasey1,Harris Thurl E.3,Snow Chelsi1,Frierson Henry F.4,Wotton David12,Paschal Bryce M.12

Affiliation:

1. Center for Cell Signaling; University of Virginia; Charlottesville Virginia

2. Department of Biochemistry and Molecular Genetics; University of Virginia; Charlottesville Virginia

3. Department of Pharmacology; University of Virginia; Charlottesville Virginia

4. Department of Pathology; University of Virginia; Charlottesville Virginia

Funder

National Cancer Institute

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

Urology,Oncology

Reference54 articles.

1. Loss of PTEN expression in paraffin-embedded primary prostate cancer correlates with high Gleason score and advanced stage;McMenamin;Cancer Res,1999

2. Frequent inactivation of PTEN/MMAC1 in primary prostate cancer;Cairns;Cancer Res,1997

3. Interfocal heterogeneity of PTEN/MMAC1 gene alterations in multiple metastatic prostate cancer tissues;Suzuki;Cancer Res,1998

4. Homozygous deletion of the PTEN tumor suppressor gene in a subset of prostate adenocarcinomas;Wang;Clin Cancer Res,1998

5. Pten is essential for embryonic development and tumour suppression;Di Cristofano;Nat Genet,1998

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