Mutated human kirstenras, driven by a thyroglobulin promoter, induces a growth advantage and partially dedifferentiates rat thyroid epithelial cellsin vitro-Reference-Cited by-同舟云学术

Mutated human kirstenras, driven by a thyroglobulin promoter, induces a growth advantage and partially dedifferentiates rat thyroid epithelial cellsin vitro

Published:1995-11-27 Issue:5 Volume:63 Page:757-760
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Monaco Carmen,Califano Daniela,Chiappetta Gennaro,Mineo Alba,De Franciscis Vittorio,Vecchio Giancarlo,Santelli Giovanni

Publisher

Wiley

Subject

Cancer Research,Oncology

Reference17 articles.

1. Cloning, chromosomal assignment, and regulation of the rat thyrotropin receptor: expression of the gene is regulated by thyrotropin, agents that increase cAMP levels, and thyroid autoantibodies.

2. Culture of hormone-dependent functional epithelial cells from rat thyroids.

3. Neoplastic transformation inactivates specific trans-acting factor(s) required for the expression of the thyroglobulin gene.

4. The sequence of 967 amino acids at the carboxyl-end of rat thyroglobulin. Location and surroundings of two thyroxine-forming sites

5. A technique for radiolabeling DNA restriction endonuclease fragments to high specific activity

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2. A minireview: the role of MAPK/ERK and PI3K/Akt pathways in thyroid follicular cell-derived neoplasm;Frontiers in Bioscience;2011

3. Oncogenic Ras Blocks the cAMP Pathway and Dedifferentiates Thyroid Cells Via an Impairment of Pax8 Transcriptional Activity;Molecular Endocrinology;2009-06

4. Forskolin, 8-Br-3′,5′-Cyclic Adenosine 5′-Monophosphate, and Catalytic Protein Kinase A Expression in the Nucleus Increase Radioiodide Uptake and Sodium/Iodide Symporter Protein Levels in RET/PTC1-Expressing Cells;The Journal of Clinical Endocrinology & Metabolism;2004-12-01

5. Genetic Alterations Involved in the Transition from Well-Differentiated to Poorly Differentiated and Anaplastic Thyroid Carcinomas;Endocrine Pathology;2004