The von Hippel-Lindau (VHL) disease tumor-suppressor gene is not mutated in nasopharyngeal carcinomas
Author:
Publisher
Wiley
Subject
Cancer Research,Oncology
Reference19 articles.
1. Establishment and characterization of three transplantable EBV-containing nasopharyngeal carcinomas
2. Germline mutations in the von Hippel-Lindau disease tumor suppressor gene: Correlations with phenotype
3. Two epithelial tumor cell lines (HNE-1 and HONE-1) latently infected with Epstein-Barr virus that were derived from nasopharyngeal carcinomas.
4. Mutations of the VHL tumour suppressor gene in renal carcinoma
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1. Limb-bud and Heart (LBH) Functions as a Tumor Suppressor of Nasopharyngeal Carcinoma by Inducing G1/S Cell Cycle Arrest;Scientific Reports;2015-01-05
2. Plasma Osteopontin, Hypoxia, and Response to Radiotherapy in Nasopharyngeal Cancer;Clinical Cancer Research;2008-11-01
3. Genetic and Epigenetic Alterations of LTF at 3p21.3 in Nasopharyngeal Carcinoma;Oncology Research Featuring Preclinical and Clinical Cancer Therapeutics;2006-06-01
4. Microsatellite Alterations as Diagnostic and Prognostic Molecular Markers in Patients With Cancer;Cancer Diagnostics;2004
5. The von Hippel-Lindau Gene and Protein in Tumorigenesis and Angiogenesis: A Potential Target for Therapeutic Designs;Current Medicinal Chemistry;2003-11-01
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