Prostaglandin E1, E2, and cholera toxin increase transcription of the brain creatine kinase gene in human U87 glioblastoma cells-Reference-Cited by-同舟云学术

Prostaglandin E1, E2, and cholera toxin increase transcription of the brain creatine kinase gene in human U87 glioblastoma cells

Published:1995-12 Issue:4 Volume:15 Page:471-479
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Kuzhikandathil Eldo V.,Molloy George R.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Reference29 articles.

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5. and (1974) Prostaglandins and the central nervous system. In: Prostaglandins: Physiological, Pharmacological and Pathological Aspects. edS. University Park Press, Baltimore, pp. 1-26.

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1. Transcription of brain creatine kinase in U87-MG glioblastoma is modulated by factor AP2;Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression;2005-04

2. Creatine and Creatinine Metabolism;Physiological Reviews;2000-07-01

3. Conditions providing enhanced transfection efficiency in rat pheochromocytoma PC12 cells permit analysis of the activity of the far-upstream and proximal promoter of the brain creatine kinase gene;Journal of Neuroscience Methods;1999-10

4. Proximal promoter of the rat brain creatine kinase gene lacks a consensus CRE element but is essential for the cAMP-mediated increased transcription in glioblastoma cells;Journal of Neuroscience Research;1999-04-27

5. Cyclooxygenase-1 and Cyclooxygenase-2 in the Human Optic Nerve Head;Experimental Eye Research;1997-12