Nrf2 in cancers: A double‐edged sword-Reference-Cited by-同舟云学术

Nrf2 in cancers: A double‐edged sword

Published:2019-03-30 Issue:5 Volume:8 Page:2252-2267
ISSN:2045-7634
Container-title:Cancer Medicine
language:en
Short-container-title:Cancer Med

Author:

Wu Shijia¹^ORCID,Lu Hong²^ORCID,Bai Yongheng¹^ORCID

Affiliation:

1. Key Laboratory of Diagnosis and Treatment of Severe Hepato‐Pancreatic Diseases of Zhejiang Province The First Affiliated Hospital of Wenzhou Medical University Wenzhou China

2. Department of Laboratory Medicine The First Affiliated Hospital of Wenzhou Medical University Wenzhou China

Publisher

Wiley

Subject

Cancer Research,Radiology, Nuclear Medicine and imaging,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cam4.2101

Reference172 articles.

1. Small Maf proteins serve as transcriptional cofactors for keratinocyte differentiation in the Keap1-Nrf2 regulatory pathway

2. Nrf2–Keap1 defines a physiologically important stress response mechanism

3. Different Electrostatic Potentials Define ETGE and DLG Motifs as Hinge and Latch in Oxidative Stress Response

4. The Carboxy-Terminal Neh3 Domain of Nrf2 Is Required for Transcriptional Activation

5. Redox-regulated Turnover of Nrf2 Is Determined by at Least Two Separate Protein Domains, the Redox-sensitive Neh2 Degron and the Redox-insensitive Neh6 Degron

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