Galectin-1 is essential for the induction of MOG35-55-based intravenous tolerance in experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

Galectin-1 is essential for the induction of MOG35-55-based intravenous tolerance in experimental autoimmune encephalomyelitis

Published:2016-05-25 Issue:7 Volume:46 Page:1783-1796
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Mari Elisabeth R.¹,Rasouli Javad¹,Ciric Bogoljub¹,Moore Jason N.¹²,Conejo-Garcia José R.³,Rajasagi Naveen⁴,Zhang Guang-Xian¹,Rabinovich Gabriel A.⁵⁶,Rostami Abdolmohamad¹

Affiliation:

1. Department of Neurology; Thomas Jefferson University; Philadelphia PA USA

2. Department of Pharmacology and Experimental Therapeutics; Thomas Jefferson University; Philadelphia PA USA

3. Tumor Microenvironment and Metastasis Program; The Wistar Institute; Philadelphia PA USA

4. Comparative and Experimental Medicine; College of Veterinary Medicine; The University of Tennessee; Knoxville TN USA

5. Laboratory of Immunopathology; Institute of Biology and Experimental Medicine (IBYME); CONICET; Buenos Aires Argentina

6. School of Exact and Natural Sciences; University of Buenos Aires; Buenos Aires Argentina

Funder

National Institutes of Health

National Institutes of Health training

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference46 articles.

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3. Autoimmune regulator: from loss of function to autoimmunity;Pitkanen;Genes Immun.,2003

4. Active suppression induced by repetitive self-epitopes protects against EAE development;Puentes;PLoS One,2013

5. Mechanisms of suppression of experimental autoimmune encephalomyelitis by intravenous administration of myelin basic protein: role of regulatory spleen cells;Hilliard;Exp. Mol. Pathol.,2000

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