Absence of nonhematopoietic MHC class II expression protects mice from experimental autoimmune myocarditis

Author:

Thelemann Christoph1,Haller Sergio1,Blyszczuk Przemyslaw2,Kania Gabriela3,Rosa Muriel1,Eriksson Urs2,Rotman Samuel4,Reith Walter5,Acha-Orbea Hans1

Affiliation:

1. Department of Biochemistry, CIIL; University of Lausanne; Epalinges Switzerland

2. Division of Cardioimmunology; Centre of Molecular Cardiology, University of Zurich; Schlieren Switzerland

3. Research of Systemic Autoimmune Diseases; Division of Rheumatology, University Hospital Zürich; Schlieren Switzerland

4. Institute of Pathology; CHUV, University of Lausanne; Lausanne Switzerland

5. Department of Pathology and Immunology; University of Geneva; Geneva Switzerland

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference45 articles.

1. Myocarditis;Feldman;N. Engl. J. Med,2000

2. Myosin-induced acute myocarditis is a T cell-mediated disease;Smith;J. Immunol.,1991

3. Identification of cardiac myosin peptides capable of inducing autoimmune myocarditis in BALB/c mice;Pummerer;J. Clin. Invest.,1996

4. Cardiac myosin induces myocarditis in genetically predisposed mice;Neu;J. Immunol,1987

5. Dendritic cell-induced autoimmune heart failure requires cooperation between adaptive and innate immunity;Eriksson;Nat. Med.,2003

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