mGluR2/3 activation of the SIRT1 axis preserves mitochondrial function in diabetic neuropathy-Reference-Cited by-同舟云学术

mGluR2/3 activation of the SIRT1 axis preserves mitochondrial function in diabetic neuropathy

Published:2017-11-01 Issue:12 Volume:4 Page:844-858
ISSN:2328-9503
Container-title:Annals of Clinical and Translational Neurology
language:en
Short-container-title:Ann Clin Transl Neurol

Author:

Chandrasekaran Krish¹²^ORCID,Muragundla Anjaneyulu¹²,Demarest Tyler G.³,Choi Joungil¹²,Sagi Avinash R.¹,Najimi Neda¹,Kumar Pranith¹^ORCID,Singh Anmol¹,Ho Cheng-Ying⁴,Fiskum Gary³,Koch Lauren G.⁵,Britton Steven L.⁵⁶,Russell James W.¹²⁷

Affiliation:

1. Department of Neurology; University of Maryland School of Medicine; Baltimore Maryland 21201

2. Veterans Affairs Medical Center; Baltimore Maryland 21201

3. Department Anesthesiology; University of Maryland School of Medicine; Baltimore Maryland 21201

4. Department Pathology; University of Maryland School of Medicine; Baltimore Maryland 21201

5. Department of Anesthesiology; University of Michigan; Ann Arbor Michigan 48109

6. Department of Molecular and Integrative Physiology; University of Michigan; Ann Arbor Michigan 48109

7. Anatomy and Neurobiology; University of Maryland School of Medicine; Baltimore Maryland 21201

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

National Institutes of Health

Office of Research and Development

U.S. Department of Veterans Affairs

Rehabilitation Research and Development Service

American Diabetes Association

Mid-Atlantic Nutrition Obesity Research Center

National Center for Research Resources

Office of Research Infrastructure Programs/OD

University of Michigan

Publisher

Wiley

Subject

Clinical Neurology,General Neuroscience

Reference55 articles.

1. Mitochondrial dysfunction in diabetic neuropathy: a series of unfortunate metabolic events;Fernyhough;Curr Diab Rep,2015

2. Dorsal root ganglia mitochondrial biochemical changes in non-diabetic and streptozotocin-induced diabetic mice fed with a standard or high-fat diet;Guilford;J Neurol Neurosci,2017

3. Heat shock protein 70 is necessary to improve mitochondrial bioenergetics and reverse diabetic sensory neuropathy following KU-32 therapy;Ma;J Pharmacol Exp Ther,2014

4. PGC-1alpha regulation of mitochondrial degeneration in experimental diabetic neuropathy;Choi;Neurobiol Dis,2014

5. Biology of diabetic neuropathy;Vincent;Handb Clin Neurol,2013

Cited by 24 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Impacts of glutamate, an exercise-responsive metabolite on insulin signaling;Life Sciences;2024-03

2. Identification and validation of NAD+ metabolism-related biomarkers in patients with diabetic peripheral neuropathy;Frontiers in Endocrinology;2024-02-23

3. NAD+ Precursors Reverse Experimental Diabetic Neuropathy in Mice;International Journal of Molecular Sciences;2024-01-16

4. BMP5 ameliorates diabetic peripheral neuropathy by augmenting mitochondrial function and inhibiting apoptosis in Schwann cells;Biochemical and Biophysical Research Communications;2023-02

5. PGC1α: an emerging therapeutic target for chemotherapy-induced peripheral neuropathy;Therapeutic Advances in Neurological Disorders;2023-01