Melatonin attenuates reactive astrogliosis and glial scar formation following cerebral ischemia and reperfusion injury mediated by GSK‐3β and RIP1K

Author:

Yawoot Nuttapong12,Sengking Jirakhamon3,Wicha Piyawadee3,Govitrapong Piyarat4,Tocharus Chainarong3,Tocharus Jiraporn15ORCID

Affiliation:

1. Department of Physiology Chiang Mai University Chiang Mai Thailand

2. Graduate School Chiang Mai University Chiang Mai Thailand

3. Department of Anatomy, Faculty of Medicine Chiang Mai University Chiang Mai Thailand

4. Chulabhorn Graduate Institute Bangkok Thailand

5. Department of Physiology, Functional Food Research Center for Well‐being Chiang Mai University Chiang Mai Thailand

Funder

Royal Golden Jubilee (RGJ) Ph.D. Programme

Chiang Mai University

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference73 articles.

1. Modulating astrocyte transition after stroke to promote brain rescue and functional recovery: Emerging targets include rho kinase;Abeysinghe H. C.;International Journal of Molecular Sciences,2016

2. Melatonin and ischemic stroke: Mechanistic roles and action;Andrabi S. S.;Advances in Pharmacological Sciences,2015

3. Intraluminal middle cerebral artery occlusion (MCAO) model for ischemic stroke with laser doppler flowmetry guidance in mice;Ansari S.;Journal of Visualized Experiments,2011

4. Melatonin regulates neuroinflammation ischemic stroke damage through interactions with microglia in reperfusion phase;Azedi F.;Brain Research,2019

5. Heart disease and stroke statistics‐2019 update: A report from the american heart association;Benjamin E. J.;Circulation,2019

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