The cytotoxic action of BCI is not dependent on its stated DUSP1 or DUSP6 targets in neuroblastoma cells
Author:
Affiliation:
1. Developmental Biology & Cancer Research and Teaching Department UCL Great Ormond Street Institute of Child Health London UK
2. Mass Spectrometry Laboratory Barts Cancer Institute Queen Mary University of London UK
Funder
Centro de Investigaciones en Optica
Neuroblastoma UK
NIHR Great Ormond Street Hospital Biomedical Research Centre
Blood Cancer UK
Publisher
Wiley
Subject
General Biochemistry, Genetics and Molecular Biology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/2211-5463.13418
Reference51 articles.
1. Neuroblastoma: biological insights into a clinical enigma
2. Neuroblastoma Origin and Therapeutic Targets for Immunotherapy
3. Children's Oncology Group's 2013 blueprint for research: Neuroblastoma
4. Advances in Risk Classification and Treatment Strategies for Neuroblastoma
5. Neuroblastoma: clinical and biological approach to risk stratification and treatment
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2. In Vitro and In Silico Investigation of BCI Anticancer Properties and Its Potential for Chemotherapy-Combined Treatments;Cancers;2023-09-06
3. Synchronisation of apical constriction and cell cycle progression is a conserved behaviour of pseudostratified neuroepithelia informed by their tissue geometry;Developmental Biology;2023-02
4. BCI, an inhibitor of the DUSP1 and DUSP6 dual specificity phosphatases, enhances P2X7 receptor expression in neuroblastoma cells;Frontiers in Cell and Developmental Biology;2022-12-15
5. Synchronisation of apical constriction and cell cycle progression is a conserved behaviour of pseudostratified neuroepithelia informed by their tissue geometry;2022-06-16
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