Megf10 deficiency impairs skeletal muscle stem cell migration and muscle regeneration-Reference-Cited by-同舟云学术

Megf10 deficiency impairs skeletal muscle stem cell migration and muscle regeneration

Published:2020-11-26 Issue:1 Volume:11 Page:114-123
ISSN:2211-5463
Container-title:FEBS Open Bio
language:en
Short-container-title:FEBS Open Bio

Author:

Li Chengcheng¹,Vargas‐Franco Dorianmarie¹,Saha Madhurima¹,Davis Rachel M.¹,Manko Kelsey A.¹,Draper Isabelle²^ORCID,Pacak Christina A.³,Kang Peter B.¹⁴⁵^ORCID

Affiliation:

1. Division of Pediatric Neurology Department of Pediatrics University of Florida College of Medicine Gainesville FL USA

2. Molecular Cardiology Research Institute Department of Medicine Tufts Medical Center Boston MA USA

3. Department of Pediatrics University of Florida College of Medicine Gainesville FL USA

4. Department of Molecular Genetics & Microbiology and Department of Neurology University of Florida College of Medicine Gainesville FL USA

5. Genetics Institute and Myology Institute University of Florida Gainesville FL USA

Funder

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

General Biochemistry, Genetics and Molecular Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/2211-5463.13031

Reference55 articles.

1. Mutations in MEGF10, a regulator of satellite cell myogenesis, cause early onset myopathy, areflexia, respiratory distress and dysphagia (EMARDD)

2. Mutations in the satellite cell gene MEGF10 cause a recessive congenital myopathy with minicores

3. Chromosomal microarray in a highly consanguineous population: diagnostic yield, utility of regions of homozygosity, and novel mutations

5. Adult-onset respiratory insufficiency, scoliosis, and distal joint hyperlaxity in patients with multiminicore disease due to novel Megf10 mutations

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