CCAL1 enhances osteoarthritis through the NF‐κB/AMPK signaling pathway
Author:
Affiliation:
1. Department of Orthopaedic Surgery Chengwu People's Hospital Heze China
2. Department of Medical College Qingdao University Qingdao China
Publisher
Wiley
Subject
General Biochemistry, Genetics and Molecular Biology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/2211-5463.12989
Reference25 articles.
1. The Role of Inflammation in the Pathogenesis of Osteoarthritis
2. Emerging targets in osteoarthritis therapy
3. Mutations in osteoprotegerin account for the CCAL1 locus in calcium pyrophosphate deposition disease
4. High levels of synovial fluid osteoprotegerin (OPG) and increased serum ratio of receptor activator of nuclear factor-κB ligand (RANKL) to OPG correlate with disease severity in patients with primary knee osteoarthritis
5. A gain of function mutation inTNFRSF11Bencoding osteoprotegerin causes osteoarthritis with chondrocalcinosis
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