Morin attenuates osteoclast formation and function by suppressing the NF‐κB , MAPK and calcium signalling pathways-Reference-Cited by-同舟云学术

Morin attenuates osteoclast formation and function by suppressing the NF‐κB , MAPK and calcium signalling pathways

Published:2021-08-22 Issue:10 Volume:35 Page:5694-5707
ISSN:0951-418X
Container-title:Phytotherapy Research
language:en
Short-container-title:Phytotherapy Research

Author:

Shi Yifeng¹²³,Ye Lin¹²³,Shen Shiwei³,Qian Tianchen³,Pan Youjin⁴,Jiang Yuhan³,Lin Jinghao³,Liu Chen³,Wu Yaosen¹²,Wang Xiangyang¹²,Xu Jiake¹²⁵,Jin Haiming¹²⁵^ORCID

Affiliation:

1. Department of Orthopaedics The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University Wenzhou China

2. Key Laboratory of Orthopaedics of Zhejiang Province Wenzhou China

3. The Second School of Medicine Wenzhou Medical University Wenzhou China

4. Department of Endocrinology The Second Affiliated Hospital and Yuying Children's Hospital, Wenzhou Medical University Wenzhou China

5. School of Biomedical Sciences The University of Western Australia Perth Western Australia Australia

Funder

National Health and Medical Research Council

Natural Science Foundation of Zhejiang Province

Zhejiang Province Public Welfare Technology Application Research Project

National Natural Science Foundation of China

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ptr.7229

Reference46 articles.

1. The molecular understanding of osteoclast differentiation

2. Morin attenuates cigarette smoke-induced lung inflammation through inhibition of PI3K/AKT/NF-κB signaling pathway

3. The emerging role of IMD 0354 on bone homeostasis by suppressing osteoclastogenesis and bone resorption, but without affecting bone formation

4. Alpinumisoflavone inhibits osteoclast differentiation and exerts anti-osteoporotic effect in ovariectomized mice

5. Chloroquine ameliorates carbon tetrachloride-induced acute liver injury in mice via the concomitant inhibition of inflammation and induction of apoptosis

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