Inflammatory arthritis in caspase 1 geneâdeficient mice: Contribution of proteinase 3 to caspase 1âindependent production of bioactive interleukin-1Î²-Reference-Cited by-同舟云学术

Inflammatory arthritis in caspase 1 geneâdeficient mice: Contribution of proteinase 3 to caspase 1âindependent production of bioactive interleukin-1Î²

Published:2009-12 Issue:12 Volume:60 Page:3651-3662
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis Rheum

Author:

Joosten Leo A. B.,Netea Mihai G.,Fantuzzi Giamila,Koenders Marije I.,Helsen Monique M. A.,Sparrer Helmut,Pham Christine T.,van der Meer Jos W. M.,Dinarello Charles A.,van den Berg Wim B.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.25006/fullpdf

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4. Interleukin-1Î² maturation and release in response to ATP and nigericin: evidence that potassium depletion mediated by these agents is a necessary and common feature of their activity;Perregaux;J Biol Chem,1994

5. Interleukin-1 receptor antagonist: a new member of the interleukin-1 family;Arend;J Clin Invest,1991

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