TRIM14 Promotes Noncanonical NF‐κB Activation by Modulating p100/p52 Stability via Selective Autophagy-Reference-Cited by-同舟云学术

TRIM14 Promotes Noncanonical NF‐κB Activation by Modulating p100/p52 Stability via Selective Autophagy

Published:2019-11-11 Issue:1 Volume:7 Page:1901261
ISSN:2198-3844
Container-title:Advanced Science
language:en
Short-container-title:Adv. Sci.

Author:

Chen Meixin¹,Zhao Zhiyao¹²,Meng Qingcai¹,Liang Puping¹,Su Zexiong¹,Wu Yaoxing¹,Huang Junjiu¹,Cui Jun¹^ORCID

Affiliation:

1. State Key Laboratory of Oncology in South ChinaMOE Key Laboratory of Gene Function and RegulationSchool of Life SciencesSun Yat‐Sen University Guangzhou Guangdong 510006 China

2. Department of Internal MedicineGuangzhou Institute of PediatricsGuangzhou Women and Children's Medical Center Guangzhou Guangdong 510623 China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

General Physics and Astronomy,General Engineering,Biochemistry, Genetics and Molecular Biology (miscellaneous),General Materials Science,General Chemical Engineering,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/advs.201901261

Reference57 articles.

1. 30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology

2. NF-κB–inducing kinase (NIK) promotes hyperglycemia and glucose intolerance in obesity by augmenting glucagon action

3. The non-canonical NF-κB pathway in immunity and inflammation

4. Shared Principles in NF-κB Signaling

5. Regulation of NF-κB by deubiquitinases

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