Functional Recovery of Contused Spinal Cord in Rat with the Injection of Optimal-Dosed Cerium Oxide Nanoparticles-Reference-Cited by-同舟云学术

Functional Recovery of Contused Spinal Cord in Rat with the Injection of Optimal-Dosed Cerium Oxide Nanoparticles

Published:2017-07-08 Issue:10 Volume:4 Page:1700034
ISSN:2198-3844
Container-title:Advanced Science
language:en
Short-container-title:Adv. Sci.

Author:

Kim Jong-Wan¹²,Mahapatra Chinmaya¹²,Hong Jin-Young²,Kim Min Soo²,Leong Kam W.¹³,Kim Hae-Won¹²⁴,Hyun Jung Keun¹²⁵

Affiliation:

1. Department of Nanobiomedical Science and BK21 PLUS NBM Global Research Center for Regenerative Medicine; Dankook University; Cheonan 330-714 Republic of Korea

2. Institute of Tissue Regeneration Engineering (ITREN); Dankook University; Cheonan 330-714 Republic of Korea

3. Department of Biomedical Engineering; Columbia University; New York NY 10027 USA

4. Department of Biomaterials Science; School of Dentistry; Dankook University; Cheonan 330-714 Republic of Korea

5. Department of Rehabilitation Medicine; College of Medicine; Dankook University; Cheonan 330-714 Republic of Korea

Funder

National Research Foundation of Korea

Korea Health Industry Development Institute

Publisher

Wiley

Subject

General Physics and Astronomy,General Engineering,Biochemistry, Genetics and Molecular Biology (miscellaneous),General Materials Science,General Chemical Engineering,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/advs.201700034/fullpdf

Reference57 articles.

1. Spinal Cord Injury Medicine. 1. Epidemiology and Classification

2. From basics to clinical: A comprehensive review on spinal cord injury

3. The paradox of chronic neuroinflammation, systemic immune suppression, autoimmunity after traumatic chronic spinal cord injury

4. Hydroxyl radicals generated in the rat spinal cord at the level produced by impact injury induce cell death by necrosis and apoptosis: protection by a metalloporphyrin

5. Reactive oxygen species contribute to neuropathic pain and locomotor dysfunction via activation of CamKII in remote segments following spinal cord contusion injury in rats

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