Mechanisms underlying the enhancement of γ‐aminobutyric acid responses in the external globus pallidus of R6/2 Huntington’s disease model mice
Author:
Affiliation:
1. IDDRC, Jane and Terry Semel Institute for Neuroscience & Human Behavior David Geffen School of Medicine University of California Los Angeles Los Angeles CA USA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24710
Reference24 articles.
1. Preferential loss of striato-external pallidal projection neurons in presymptomatic Huntington's disease
2. Cannabinoid (CB1), GABAA and GABAB receptor subunit changes in the globus pallidus in Huntington's disease
3. Striatal Direct and Indirect Pathway Output Structures Are Differentially Altered in Mouse Models of Huntington's Disease
4. Multiple Sources of Striatal Inhibition Are Differentially Affected in Huntington's Disease Mouse Models
5. The role of dopamine in huntington's disease
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