Dexamethasone inhibits feedback regulation of the mitogenic activity of tumor necrosis factor, interleukin-1, and epidermal growth factor in human fibroblasts-Reference-Cited by-同舟云学术

Dexamethasone inhibits feedback regulation of the mitogenic activity of tumor necrosis factor, interleukin-1, and epidermal growth factor in human fibroblasts

Published:1987-08 Issue:2 Volume:132 Page:271-278
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J. Cell. Physiol.

Author:

Kohase Masayoshi,Henriksen-Destefano Dorothy,Sehgal Pravinkumar B.,Vil?ek Jan

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference47 articles.

1. Dexamethasone modulates binding and action of epidermal growth factor in serum-free cell culture

2. Mitogenesis in normal human fibroblasts by polyinosinic · polycytidylic acid and other synthetic acidic polymers: Enhancement of action by glucocorticoids

3. Macrocortin: a polypeptide causing the anti-phospholipase effect of glucocorticoids

4. Purification and Biochemical Characterization of the Promoter-Specific Transcription Factor, Sp1

5. Dexamethasone and retinyl acetate similarly inhibit and stimulate EGF-or insulin-induced proliferation of prostatic epithelium

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