Proteoglycan biosynthesis is required in BC3H1 myogenic cells for modulation of vascular smooth muscle ?-actin gene expression in response to microenvironmental signals-Reference-Cited by-同舟云学术

Proteoglycan biosynthesis is required in BC3H1 myogenic cells for modulation of vascular smooth muscle ?-actin gene expression in response to microenvironmental signals

Published:1995-07 Issue:1 Volume:164 Page:172-186
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J. Cell. Physiol.

Author:

Lee Sang Hoon,Yan Hua,Reeser Jonathan C.,Dillman John M.,Strauch Arthur R.

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference73 articles.

1. Regulation of development and differentiation by the extracellular matrix

2. and (1993) Current Protocols in Molecular Biology, John, Wiley and Sons, New York.

3. Coexpression of α-sarcomeric actin, α-smooth muscle actin and desmin during myogenesis in rat and mouse embryos I. Skeletal muscle

4. Differential expression of SM1 and SM2 myosin isoforms in cultured vascular smooth muscle

5. (1987) Smooth muscle cell attachment and differentiation: a model system. M.S. Thesis, The Ohio State University.

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