M2 subunit of ribonucleotide reductase is a target of cyclic AMP-dependent protein kinase
Author:
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Reference31 articles.
1. Overproduction of the free radical of ribonucleotide reductase in hydroxyurea-resistant mouse fibroblast 3T6 cells.
2. Ribonucleotide reductase activity and deoxyribonucleoside triphosphate metabolism during the cell cycle of S49 wild-type and mutant mouse T-lymphoma cells.
3. Deoxyribonucleotide metabolism and cyclic AMP resistance in hydroxyurea-resistant S49 T-lymphoma cells
4. Gene for M1 subunit of ribonucleotide reductase is amplified in hydroxyurea-resistant hamster cells
5. Cyclic AMP, a nonessential regulator of the cell cycle.
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1. Malignant transformation by H-ras results in aberrant regulation of ribonucleotide reductase gene expression by transforming growth factor-β1;Journal of Cellular Biochemistry;1995-03
2. CGRP in the Serum of Endotoxin-Treated Rats Suppresses Lymphoproliferation;Brain, Behavior, and Immunity;1994-12
3. Phosphorylation of ribonucleotide reductase R2 protein: In vivo and in vitro evidence of a role for p34cdc2 and CDK2 protein kinases;Biochemistry;1993-11-01
4. Ribonucleotide reductase gene expression during cyclic AMP-induced cell cycle arrest in T lymphocytes;Experimental Cell Research;1992-12
5. Beta-adrenergic, cAMP-mediated stimulation of proliferation of brown fat cells in primary culture. Mediation via beta 1 but not via beta 3 adrenoceptors.;Journal of Biological Chemistry;1992-01
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