Promotion of Calcium/Calmodulin‐Dependent Protein Kinase 4 by GLUT1‐Dependent Glycolysis in Systemic Lupus Erythematosus
Author:
Affiliation:
1. Nagasaki University Graduate School of Biomedical Sciences Nagasaki Japan
2. Nagasaki University Graduate School of Biomedical Sciences and Nagasaki University Hospital Nagasaki Japan
Funder
Japan Society for the Promotion of Science
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.40785
Reference17 articles.
1. Cellular and Molecular Mechanisms of Regulation of Autoantibody Production in Lupus
2. Deficient CD4+CD25high T Regulatory Cell Function in Patients with Active Systemic Lupus Erythematosus
3. HIF1α–dependent glycolytic pathway orchestrates a metabolic checkpoint for the differentiation of TH17 and Treg cells
4. Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways
5. The Glucose Transporter Glut1 Is Selectively Essential for CD4 T Cell Activation and Effector Function
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