Brusatol overcomes chemoresistance through inhibition of protein translation-Reference-Cited by-同舟云学术

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Brusatol overcomes chemoresistance through inhibition of protein translation

Published:2017-02-08 Issue:5 Volume:56 Page:1493-1500
ISSN:0899-1987
Container-title:Molecular Carcinogenesis
language:en
Short-container-title:Mol. Carcinog.

Author:

Harder Bryan¹,Tian Wang¹,La Clair James J.¹,Tan Aik-Choon²,Ooi Aikseng¹,Chapman Eli¹,Zhang Donna D.¹³

Affiliation:

1. Department of Pharmacology and Toxicology; University of Arizona; Tucson Arizona

2. Division of Medical Oncology. School of Medicine; University of Colorado Anschutz Medical Campus; Aurora Colorado

3. University of Arizona Cancer Center (UACC); University of Arizona; Tucson Arizona

Funder

National Cancer Institute

Publisher

Wiley

Subject

Cancer Research,Molecular Biology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/mc.22609/fullpdf

Reference29 articles.

1. Targeted therapy for non-small cell lung cancer: Current standards and the promise of the future;Chan;Transl Lung Cancer Res,2015

2. Nrf2 enhances resistance of cancer cells to chemotherapeutic drugs, the dark side of Nrf2;Wang;Carcinogenesis,2008

3. The emerging role of the Nrf2-Keap1 signaling pathway in cancer;Jaramillo;Genes Dev,2013

4. Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2;Kobayashi;Mol Cell Biol,2004

5. Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex;Zhang;Mol Cell Biol,2004

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