Resolvin E1 Improves Mitochondrial Function in Human Alveolar Epithelial Cells during Severe Inflammation

Author:

Mayer Konstantin1,Sommer Natascha1,Hache Karl1,Hecker Andreas2,Reiche Sylvia1,Schneck Emmanuel3,Weissmann Norbert1,Seeger Werner14,Hecker Matthias1

Affiliation:

1. Medical Clinic II, University of Giessen and Marburg Lung Center (UGMLC); Justus-Liebig-University; Klinikstr. 33, 35392 Giessen Germany

2. Department of General and Thoracic Surgery; University Hospital of Giessen, Rudolf-Buchheim-Str. 7; 35392 Giessen Germany

3. Department of Anesthesiology and Intensive Care Medicine; University Hospital of Giessen, Rudolf-Buchheim-Str. 7; 35392 Giessen Germany

4. Max Planck Institute for Heart and Lung Research, Department of Lung Development and Remodelling, Ludwigstr. 43; 61231 Bad Nauheim Germany

Publisher

Wiley

Subject

Cell Biology,Organic Chemistry,Biochemistry

Reference55 articles.

1. Stereochemical assignment, antiinflammatory properties, and receptor for the omega-3 lipid mediator resolvin E1;Arita;Journal of Experimental Medicine,2005a

2. Resolvin E1 selectively interacts with leukotriene B4 receptor BLT1 and ChemR23 to regulate inflammation;Arita;Journal of Immunology,2007

3. Resolvin E1, an endogenous lipid mediator derived from omega-3 eicosapentaenoic acid, protects against 2,4,6-trinitrobenzene sulfonic acid-induced colitis;Arita;Proceedings of the National Academy of Sciences of the United States of America,2005b

4. Metabolic stress and disorders related to alterations in mitochondrial fission or fusion;Babbar;Molecular and Cellular Pharmacology,2013

5. Quantitative PCR analysis of mitochondrial DNA content in patients with mitochondrial disease;Bai;Annals of the New York Academy of Sciences,2004

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